By J D Pickard; et al

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Studies using fMRI . . . . . . . . . . . . . . . . . . . . . . . . . . . Brain responses to pain . . . . . . . . . . . . . . . . . . . . . . . . J. D. Pickard et al. ), Advances and Technical Standards in Neurosurgery © Springer-Verlag/Wien 2011 26 27 27 27 27 28 29 30 31 31 32 35 35 36 42 42 42 42 43 26 J. MAARRAWI et al. Opioid receptor studies using PET . . . . . . . . . . . . . . . . . . . . Opioid receptor availability changes in neuropathic pain .

In: Garland B (ed) Neuroscience and the law: brain, mind and the scales of justice. Dana Press, New York, pp 157–98 59. Nachev P, Kennard C, Husain M (2008) Functional role of the supplementary and presupplemenatary motor areas. Nat Rev Neurosci 9: 856–69 60. Nahmias E, Morris SG, Nadelhofer T, Turner J (2006) Is incompatibilism intuitive? Philos Phenomen Res 73: 28–52 61. Nichols S, Knobe J (2007) Moral responsibility and determinism: The cognitive science of folk intuitions. No^us 41: 663–85 62.

Brain stem lesions In cases of dissociated sensory loss due to brain stem disease affecting the spinothalamic pathways, LEPs are abnormal while SEPs remain within normal limits [63, 83, 128, 163]. LEP abnormalities are mainly represented by the absence of response or amplitude attenuation and only rarely by latency delays. In the case of reversible lesions, such as brain stem encephalitis, LEPs have demonstrated their ability to follow the recovery of spinothalamic transmission [63]. LEPs to trigeminal stimulation have been found abnormal in brain stem infarctions and brain stem demyelinating plaques affecting the spinothalamic pathways, including patients in whom brain stem reflexes remained normal [123].

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